Practice MCQs for NEET Biology Chemical Coordination and Integration
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Multiple Choice Questions
(a) Melatonin Sleep wake cycle and body temperature
(b) FSH Growth of ovarian follicles and stimulate gonadal activity.
(c) Adrenaline Increase concentration of glucose in blood.
(d) Progesterone Stimulate growth and activities of female secondary sex organs
Show Answer & Explanation
Answer: (d) Progesterone Stimulate growth and activities of female secondary sex organs
Explanation:
1. Progesterone prepares and maintains the uterus for pregnancy; estrogen stimulates growth and activity of female secondary sex organs.
2. Therefore the match in option 4 is wrong.
Teacher's Note:
1. Estrogen: secondary sexual characters.
2. Progesterone: pregnancy.
(a) Insulin and glucagon
(b) Adrenalin and glucagon
(c) Adrenalin and Insulin
(d) Glucagon and growth hormone
Show Answer & Explanation
Answer: (c) Adrenalin and Insulin
Explanation:
1. Adrenaline raises blood glucose (hyperglycaemic) and insulin lowers it (hypoglycaemic).
2. So adrenaline and insulin are the hyperglycaemic and hypoglycaemic pair respectively.
Teacher's Note:
1. Hypoglycaemic: insulin only.
2. Hyperglycaemic: glucagon, adrenaline, cortisol.
(a) a = oxytocin, c = portal circulation; b = ADH, d = direct release
(b) a = ADH, c = axonal transport; b = TSH-releasing hormone, d = portal circulation
(c) a = ACTH, c = axonal transport; b = MSH, d = portal circulation
(d) a = TSH-releasing hormone, c = axonal transport; b = ADH, d = portal circulation
Show Answer & Explanation
Answer: (b) a = ADH, c = axonal transport; b = TSH-releasing hormone, d = portal circulation
Explanation:
1. ADH is synthesised in the hypothalamus and reaches the posterior pituitary by axonal transport.
2. Releasing hormones such as TSH-RH reach the anterior pituitary through the portal circulation.
Teacher's Note:
1. Neural route: posterior lobe.
2. Portal route: anterior lobe.
(a) a, c
(b) b, c
(c) c, d
(d) b, d
Show Answer & Explanation
Answer: (d) b, d
Explanation:
1. The thymus is linked to myasthenia gravis and the pancreas to prolonged hyperglycaemia (diabetes).
2. The pineal gland does not control metabolism and mental retardation, and the thyroid is not anti-inflammatory.
Teacher's Note:
1. Cortisol gives anti-inflammatory action.
2. Thymectomy relieves myasthenia gravis.
(a) Libido
(b) Growth of facial hair
(c) High pitch voice
(d) Pregnancy support
Show Answer & Explanation
Answer: (d) Pregnancy support
Explanation:
1. The ruptured follicle becomes the corpus luteum, which secretes progesterone for pregnancy.
2. Libido, facial hair and voice depend on androgens.
Teacher's Note:
1. Progesterone supports pregnancy.
2. Estrogen helps female characters.
(a) ANF – Heart, Calcitonin – parathyroid
(b) Renin – Kidney, Relaxin – Placenta
(c) Calcitonin – Kidney, HCG ® Ovary
(d) Oestrogen – Testes, Progesteron® Graafian follicle
Show Answer & Explanation
Answer: (b) Renin – Kidney, Relaxin – Placenta
Explanation:
1. Renin is secreted by the kidney and relaxin by the placenta (and ovary).
2. Calcitonin is from the thyroid, HCG from the placenta, and estrogen from ovary.
Teacher's Note:
1. ANF: atria.
2. Calcitonin: thyroid.
(a) Hypercalcemia
(b) Hyperglycemia
(c) Hyperkalemia
(d) Hypocalcemia and hypoglycemia both
Show Answer & Explanation
Answer: (a) Hypercalcemia
Explanation:
1. Parathormone raises blood calcium, causing hypercalcaemia when in excess.
2. It does not lower calcium.
Teacher's Note:
1. PTH is hypercalcaemic.
2. Calcitonin is hypocalcaemic.
(a) Adrenal cortex
(b) Thymus
(c) Thyroid
(d) Pituitary
Show Answer & Explanation
Answer: (c) Thyroid
Explanation:
1. Thyroid hormones regulate BMR and body temperature.
2. Adrenal cortex, thymus and pituitary do not do this directly.
Teacher's Note:
1. T3 and T4: calorigenic.
2. Hypothyroidism: cold intolerance.
(a) Prostaglondins
(b) Aldosterone
(c) Estrogen
(d) Cortisol
Show Answer & Explanation
Answer: (b) Aldosterone
Explanation:
1. Aldosterone increases Na+ reabsorption and K+ excretion in the distal tubule.
2. Cortisol and prostaglandins do not do this mainly.
Teacher's Note:
1. Aldosterone: mineralocorticoid.
2. Controls electrolytes.
(a) Increased blood calcium level
(b) Decreased blood calcium level
(c) Both 1 and 2
(d) Increased blood sugar level.
Show Answer & Explanation
Answer: (a) Increased blood calcium level
Explanation:
1. Thyrocalcitonin is released when blood calcium rises.
2. It lowers calcium by reducing bone resorption.
Teacher's Note:
1. Rise in Ca2+ triggers TCT.
2. Fall in Ca2+ triggers PTH.
(a) TCT
(b) Catecholamines
(c) Cortisol
(d) Thymosins
Show Answer & Explanation
Answer: (b) Catecholamines
Explanation:
1. Catecholamines (adrenaline and noradrenaline) increase alertness, piloerection and sweating.
2. Cortisol and thymosin do not act this way.
Teacher's Note:
1. Medullary hormones.
2. Fast acting.
(a) Development of embryo becomes abnormal
(b) Abortion occurs after some time
(c) Embryo develops normally till birth
(d) None
Show Answer & Explanation
Answer: (c) Embryo develops normally till birth
Explanation:
1. From the second trimester the placenta produces enough progesterone.
2. So removal of the ovaries in the fourth month does not stop development.
Teacher's Note:
1. Placenta: endocrine organ.
2. Early pregnancy needs corpus luteum.
(a) Rickets and mongolism
(b) Diabetes mellitus and cretinism
(c) Gigantism and exophthalmia
(d) Cretinism and diabetes
Show Answer & Explanation
Answer: (a) Rickets and mongolism
Explanation:
1. Rickets is a vitamin D deficiency disease and mongolism is a chromosomal disorder, not hormonal.
2. The other pairs involve insulin, thyroxine or growth hormone.
Teacher's Note:
1. Down syndrome: trisomy 21.
2. Rickets: vitamin D.
(a) Relaxin
(b) HCG
(c) Placental lactogen
(d) Estradiol
Show Answer & Explanation
Answer: (d) Estradiol
Explanation:
1. Relaxin, HCG and placental lactogen are protein hormones.
2. Estradiol is a steroid.
Teacher's Note:
1. Steroid: estradiol.
2. Protein: HCG.
(a) Pituitary - FSH
(b) Thyroid - T4
(c) Ovary - Calcitonin
(d) Islets of Langerhans - insulin
Show Answer & Explanation
Answer: (c) Ovary - Calcitonin
Explanation:
1. The ovary secretes estrogen and progesterone; calcitonin is made by the thyroid.
2. Therefore ovary-calcitonin is incorrect.
Teacher's Note:
1. Calcitonin: thyroid.
2. Inhibin: gonads.
(a) Pituitary gland
(b) pineal body
(c) Lens of eye
(d) All of the above
Show Answer & Explanation
Answer: (d) All of the above
Explanation:
1. Pituitary (adenohypophysis from Rathke's pouch, neurohypophysis from the brain), pineal and the lens of the eye all arise from ectoderm.
2. So all are ectodermal.
Teacher's Note:
1. Ectodermal derivatives.
2. Neural origin of pineal.
(a) Thymus
(b) Parathyroid
(c) Adrenal Cortex
(d) Thyroid
Show Answer & Explanation
Answer: (c) Adrenal Cortex
Explanation:
1. The adrenal cortex arises from mesoderm.
2. Thymus, parathyroid and thyroid arise from endoderm.
Teacher's Note:
1. Cortex: mesoderm.
2. Medulla: ectoderm.
(a) Relaxin – Gigantism
(b) Prolactin – Cretinism
(c) Parathyroid hormone – Tetany
(d) Insulin – Diabetes insipidus
Show Answer & Explanation
Answer: (c) Parathyroid hormone – Tetany
Explanation:
1. Deficiency of parathyroid hormone lowers blood calcium and causes tetany.
2. Relaxin does not cause gigantism, prolactin does not cause cretinism and insulin deficiency causes diabetes mellitus.
Teacher's Note:
1. PTH deficit: tetany.
2. Insulin deficit: DM.
(a) Insulin – Diabetes insipidus
(b) Thyroxine – Tetany
(c) Parathyroid hormone – Diabetes mellitus
(d) Luteinizing hormone–Failure of ovulation
Show Answer & Explanation
Answer: (d) Luteinizing hormone–Failure of ovulation
Explanation:
1. LH is needed for ovulation, so its deficiency causes failure of ovulation.
2. Insulin deficiency gives diabetes mellitus, thyroxine deficiency gives cretinism or myxoedema.
Teacher's Note:
1. LH: ovulation.
2. PTH: calcium.
(a) Cushing's disease
(b) Conn's disease
(c) Addison's disease
(d) Grave's disease
Show Answer & Explanation
Answer: (b) Conn's disease
Explanation:
1. Conn's disease is primary hyperaldosteronism, excess mineralocorticoid independent of the renin-angiotensin system.
2. Cushing's is glucocorticoid excess.
Teacher's Note:
1. Conn: aldosterone excess.
2. Addison: deficiency.
(a) CAMP
(b) CGMP
(c) GTP
(d) ATP
Show Answer & Explanation
Answer: (a) CAMP
Explanation:
1. cAMP is the second messenger that mediates the action of adrenaline in heart muscle.
2. cGMP often has the opposite effect.
Teacher's Note:
1. cAMP speeds contraction.
2. cGMP slows contraction.
(a) Gynosism
(b) Gynochorism
(c) Feminization
(d) Gynaecomastia
Show Answer & Explanation
Answer: (d) Gynaecomastia
Explanation:
1. Enlargement of breasts in males is gynaecomastia.
2. The other names are not valid terms.
Teacher's Note:
1. Gynaecomastia: male breast enlargement.
2. Linked to estrogen excess.
Chemical Coordination and Integration Objective Questions & Solutions for NEET Biology
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FAQs
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